College of Medicine-Tucson
Professor, Medical Imaging, Professor, Biomedical Engineering, Member of the Graduate Faculty
My lab develops magnetic resonance imaging (MRI) techniques with an emphasis on quantitative imaging to improve the diagnosis of early disease and the effect of treatment. Main areas of disease focus are cancer, cardiovascular, and metabolic disorders. A focus of our work is clinical translation, thus the methods that we developed provide high quality and accurate quantitative imaging within the time constraints of a clinical MRI scan. Support for our work is provided by the National Institutes of Health, the Arizona Biomedical research Centre, the American Heart Association, and industry and our technology is distributed worldwide.
Assistant Professor, Physiology , Assistant Professor, Surgery , Member of the General Faculty, Member of the Graduate Faculty
Dr. Pires is an Assistant Professor and Principal Investigator in the Department of Physiology, University of Arizona College of Medicine Tucson. Dr. Pires received his Ph.D. in Pharmacology and Toxicology at Michigan State University and completed his training as a Postdoctoral Fellow at the University of Nevada, Reno School of Medicine. Throughout his career Dr. Pires has published numerous research articles on the impact of chronic cardiovascular diseases in development of cerebral vascular disorders, such as ischemic strokes, as well as mechanisms regulating cerebral vascular function. In his laboratory, Dr. Pires' research focuses on the vascular component underlying neurodegenerative diseases, such as cerebral amyloid angiopathy and Alzheimer's diseases, as well as the brain waste clearance system, the glymphatic / cervical lymphatic system.
Associate Professor, Medicine , Member of the General Faculty, Member of the Graduate Faculty, Associate Professor, BIO5 Institute
Dr. Rafikov laboratory is focused on molecular mechanisms of pulmonary vascular cell dysfunction in pulmonary hypertension and acute lung injury. The lab is exploring mechanisms of metabolic reprogramming, mitochondrial dysfunction, and proliferative signaling.