Stefano Guerra

Photo of Stefano Guerra

Stefano Guerra

Director, Epidemiology
Professor, Public Health
Professor, Medicine - (Tenure Track)
Research Scientist, Respiratory Sciences
Professor, BIO5 Institute
Primary Department
Asthma & Airway Disease Research Center
Department Affiliations
Asthma & Airway Disease Research Center
BIO5 Institute
Contact
(520) 626-7411
stefano@arizona.edu

Work Summary

Stefano Guerra's work includes an epidemiologic study, which used a household-based approach to assess prevalence and longitudinal changes in respiratory health. Other biomarker projects include a study on molecular biomarkers of asthma and COPD from the European Community Respiratory Health Survey.

Research Interest

Stefano Guerra, MD, PhD, is a professor of Medicine, the Director of the Population Science Unit at the Asthma and Airway Disease Research Center, and a leading expert in the natural history and biomarkers of obstructive lung diseases, including asthma and chronic obstructive pulmonary disease (COPD). As principal investigator, he is engaged in the leadership and coordination of multiple studies that use bio-specimens and phenotypic information from independent epidemiological cohorts to characterize the natural history, profile the risk factors, and identify novel biomarkers of lung diseases.

Publications

Kubesch, N. J., de Nazelle, A., Westerdahl, D., Martinez, D., Carrasco-Turigas, G., Bouso, L., Guerra, S., & Nieuwenhuijsen, M. J. (2015). Respiratory and inflammatory responses to short-term exposure to traffic-related air pollution with and without moderate physical activity. Occupational and environmental medicine, 72(4), 284-93.

Exposure to traffic-related air pollution (TRAP) has been associated with adverse respiratory and systemic outcomes. Physical activity (PA) in polluted air may increase pollutant uptake and thereby health effects. The authors aimed to determine the short-term health effects of TRAP in healthy participants and any possible modifying effect of PA.

Guerra, S., Halonen, M., Vasquez, M. M., Spangenberg, A., Stern, D. A., Morgan, W. J., Wright, A. L., Lavi, I., Tarès, L., Carsin, A., Dobaño, C., Barreiro, E., Zock, J., Martínez-Moratalla, J., Urrutia, I., Sunyer, J., Keidel, D., Imboden, M., Probst-Hensch, N., , Hallberg, J., et al. (2015). Relation between circulating CC16 concentrations, lung function, and development of chronic obstructive pulmonary disease across the lifespan: a prospective study. The Lancet. Respiratory medicine, 3(8), 613-20.
BIO5 Collaborators
Stefano Guerra, Fernando Martinez

Low concentrations of the anti-inflammatory protein CC16 (approved symbol SCGB1A1) in serum have been associated with accelerated decline in forced expiratory volume in 1 s (FEV1) in patients with chronic obstructive pulmonary disease (COPD). We investigated whether low circulating CC16 concentrations precede lung function deficits and incidence of COPD in the general population.

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